JCM Figure table search 04
Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Cappelli, E. A.
Right arrow Articles by Merquior, V. L. C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Cappelli, E. A.
Right arrow Articles by Merquior, V. L. C.

 Previous Article  |  Next Article 

Journal of Clinical Microbiology, December 1999, p. 4124-4126, Vol. 37, No. 12
0095-1137/99/$04.00+0
Copyright © 1999, American Society for Microbiology. All rights reserved.

Leuconostoc pseudomesenteroides as a Cause of Nosocomial Urinary Tract Infections

Elisabete A. Cappelli,1 Rosana R. Barros,2 Thereza Cristina F. Camello,3 Lúcia M. Teixeira,2 and Vânia Lúcia C. Merquior1,*

Faculdade de Ciências Médicas1 and Hospital Universitário Pedro Ernesto,3 Universidade do Estado do Rio de Janeiro, Rio de Janeiro 20551, and Instituto de Microbiologia Prof. Paulo de Goes, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941,2 Brazil

Received 9 December 1998/Returned for modification 1 June 1999/Accepted 9 August 1999

The phenotypic and genotypic characterization of five clinical isolates of Leuconostoc pseudomesenteroides associated with nosocomially acquired urinary tract infections is described. All the strains were susceptible to chloramphenicol, clindamycin, erythromycin, gentamicin, and tetracycline; all were resistant to nalidixic acid, norfloxacin, and vancomycin; and all were intermediately affected by ampicillin and penicillin. Analysis of chromosomal DNA by pulsed-field gel electrophoresis after treatment with SmaI indicated a clonal relationship of the isolates. The results provide evidence for the possibility of nosocomial transmission of this unusual opportunistic, vancomycin-resistant pathogen.

* Corresponding author. Mailing address: Disciplina de Microbiologia e Imunologia, Faculdade de Ciências Médicas, Universidade do Estado do Rio de Janeiro, Av. 28 de Setembro, 87-fds / 3° andar Vila Isabel, 20551 Rio de Janeiro, RJ, Brazil. Phone: 55 21 587 6380. Fax: 55 21 587 6476. E-mail: merquior{at}uerj.br.

Journal of Clinical Microbiology, December 1999, p. 4124-4126, Vol. 37, No. 12
0095-1137/99/$04.00+0
Copyright © 1999, American Society for Microbiology. All rights reserved.


This article has been cited by other articles:



Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
Antimicrob. Agents Chemother. Clin. Microbiol. Rev.
Clin. Vaccine Immunol. ALL ASM JOURNALS

Copyright © 1999 by the American Society for Microbiology. All rights reserved.