| |||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||
Previous Article | Next Article ![]()
Journal of Clinical Microbiology, January 2008, p. 338-340, Vol. 46, No. 1
0095-1137/08/$08.00+0 doi:10.1128/JCM.01177-07
Copyright © 2008, American Society for Microbiology. All Rights Reserved.

Department of Medical Parasitology,1 Department of Hospital Hygiene, Clinical Institute of Hygiene and Medical Microbiology, Medical University of Vienna, Vienna, Austria,3 2nd Medical Department, Pulmonological Centre SMZ Baumgartner Höhe, Otto Wagner-Hospital, Vienna, Austria,2 Division of Parasitic Diseases, Centers for Disease Control and Prevention, Atlanta, Georgia4
Received 12 June 2007/ Returned for modification 18 October 2007/ Accepted 31 October 2007
Acanthamoeba amoebae of genotype T2 were identified as the causative agent of Acanthamoeba skin lesions and granulomatous amoebic encephalitis (GAE) in a human immunodeficiency virus-negative patient with underlying tuberculosis. To our knowledge this, is the first case of GAE involving genotype T2.
Published ahead of print on 14 November 2007.
| Antimicrob. Agents Chemother. | Clin. Microbiol. Rev. |
|---|---|
| Clin. Vaccine Immunol. | ALL ASM JOURNALS |
|---|